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MOTS-c 10 mg pen

Mitochondrial, pen

MOTS-c

16-amino-acid mitochondrial-derived peptide. Activates AMPK; studied for metabolic homeostasis and energy regulation.

€64.90

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10 mg is sold out right now. Stock is checked hourly.

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Sold and shipped by Purita Peptides, the seller of record. You pay on their secure checkout.
In the box
1 x Pre-Formulated Peptide Pen
Format
Pen
Storage
Keep refrigerated at 2°C to 8°C. Do not freeze. Keep away from direct light.
Delivery
Worldwide, tracked and insured, 2 to 3 business days after dispatch. Dispatched within 1 to 2 business days of payment. Shipping details
Certificate
None published for this product yet. See what is available
Use
For laboratory research only. Not for human or animal use.

MOTS-c, 10 mg

€64.90

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Specification

Molecular weight
2,174.46 Da
Half-life
30 min
Research area
Research / other
Format
Pen

Also called Mitochondrial-Derived Peptide

~30 min plasma t½ (rodent, SC). No published human PK. Native peptide — no albumin tether or PEG. Muscle/tissue retention persists ~6–8 h after plasma clearance.

Sequence M R W Q E M G Y I F Y P R K L R

Certificate of analysis

Purita Peptides has not published a certificate for MOTS-c yet. We list a certificate only once the document exists and we have opened it. See which products have certificates.

About the research

MOTS-c (Mitochondrial Open Reading Frame of the 12S rRNA-c) is one of the first identified mitochondrial-derived peptides. It is encoded within the mitochondrial genome rather than the nuclear genome, which makes it an unusual subject in research on mitochondrial-nuclear retrograde signaling and metabolic adaptation.

Mechanism, reported research areas, literature dose ranges and co-factor studies are on the research page, with sources. Nothing there is a claim about this product.

Read the MOTS-c research overview
  • AMPK pathway research
  • Metabolic homeostasis studies
  • Mitochondrial signaling research
  • Insulin sensitivity models
  • Reported research areas, from the literature. Not product claims.

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